References

Bennett M Vitamin B12 deficiency, infertility and recurrent fetal loss. J Reprod Med. 2001; 46:(3)209-12

BOC Healthcare. 2015. http://tinyurl.com/jetzfry (accessed 24 October 2016)

BOC Healthcare. 2016. http://tinyurl.com/jmh3m35 (accessed 24 October 2016)

de Jager J, Kooy A, Lehert P, Wulffelé MG, van der Kolk J, Bets D, Verburg J, Donker AJM, Stehouwer CDA Long term treatment with metformin in patients with type 2 diabetes and risk of vitamin B-12 deficiency: randomised placebo controlled trial. BMJ. 2010; 2010 https://doi.org/http://dx.doi.org/10.1136/bmj.c2181

Devalia V, Hamilton MS, Molloy AM Guidelines for the diagnosis and treatment of cobalamin and folate disorders. Br J Haematol. 2014; 166:(4)496-513 https://doi.org/10.1111/bjh.12959

Dobson R, Alvares D The difficulties with vitamin B12. Pract Neurol. 2016; 16:(4)308-11 https://doi.org/10.1136/practneurol-2015-001344

Exclusive breastfeeding may cause B12 deficiency in babies. 2013. http://tinyurl.com/jg4n2yq (accessed 24 October 2016)

Guez S, Chiarelli G, Menni F, Salera S, Principi N, Esposito S Severe vitamin B12 deficiency in an exclusively breastfed 5-month-old Italian infant born to a mother receiving multivitamin supplementation during pregnancy. BMC Pediatr. 2012; 12 https://doi.org/10.1186/1471-2431-12-85

Hector M, Burton JR What are the psychiatric manifestations of vitamin B12 deficiency?. J Am Geriatr Soc. 1988; 36:(12)1105-12

Herrmann W, Obeid R Causes and early diagnosis of vitamin B12 deficiency. Dtsch Arztebl Int. 2008; 105:(40)680-5 https://doi.org/10.3238/arztebl.2008.0680

Hunt A, Harrington D, Robinson S Vitamin B12 deficiency. BMJ. 2014; 349 https://doi.org/10.1136/bmj.g5226

Prevention of anencephaly. 2015. http://www.anencephaly.info/e/prevention.php (accessed 24 October 2016)

Vitamin B12 deficiency and pernicious anaemia. 2016. http://patient.info/health/vitamin-b12-deficiency-and-pernicious-anaemia (accessed 24 October 2016)

Lindenbaum J, Healton EB, Savage DG, Brust JC, Garrett TJ, Podell ER, Marcell PD, Stabler SP, Allen RH Neuropsychiatric disorders caused by cobalamin deficiency in the absence of anemia or macrocytosis. N Engl J Med. 1988; 318:(26)1720-8

National Institute for Health and Care Excellence. Active B12 assay for diagnosing vitamin B12 deficiency. 2015a. http://www.nice.org.uk/advice/mib40 (accessed 24 October 2016)

National Institute for Health and Care Excellence. Diabetes in pregnancy: management from preconception to the postnatal period. 2015b. http://www.nice.org.uk/guidance/ng3 (accessed 24 October 2016)

NHS Choices. Vitamins and minerals – B vitamins and folic acid. 2015. http://www.nhs.uk/Conditions/vitamins-minerals/Pages/Vitamin-B.aspx (accessed 24 October 2016)

NHS Choices. Vitamin B12 or folate deficiency anaemia. 2016. http://www.nhs.uk/conditions/Anaemia-vitamin-B12-and-folate-deficiency/Pages/Introduction.aspx (accessed 24 October 2016)

Pacholok S, Stuart J, 2nd edn. Fresno, CA: Quill Driver Books; 2011

Piziali RL, Whitcher C, Sher R, Moffat RJ Distribution of waste anesthetic gases in the operating room air. Anesthesiology. 1976; 45:(5)487-94

Pepper MR, Black MM B12 in fetal development. Semin Cell Dev Biol. 2011; 22:(6)619-23 https://doi.org/10.1016/j.semcdb.2011.05.005

Royal College of Midwives. RCM Entonox Guidance. 2016. http://www.rcm.org.uk/content/entonox (accessed 24 October 2016)

Sahoo MK, Avasthi A, Singh P Negative symptoms presenting as neuropsychiatric manifestation of vitamin B12 deficiency. Indian J Psychiatry. 2011; 53:(4)370-1 https://doi.org/10.4103/0019-5545.91914

Savage DG, Lindenbaum J Neurological complications of acquired cobalamin deficiency: clinical aspects. Baillieres Clin Haematol. 1995; 8:(3)657-78

Vitamin B12 associated neurological diseases. 2016. http://emedicine.medscape.com/article/1152670-overview (accessed 24 October 2016)

Stichting Tekort. Treatment with high dose vitamin B12 been shown to be safe for more than 50 years. 2016. http://tinyurl.com/j3o7rfs (accessed 24 October 2016)

What Every Vegan Should Know about vitamin B12. 2001. http://tinyurl.com/zeuv76b (accessed 24 October 2016)

World Health Organization. Micronutrient deficiencies. 2016. http://www.who.int/nutrition/topics/ida/ (accessed 24 October 2016)

Vitamin B12 deficiency

02 November 2016
Volume 24 · Issue 11

Abstract

Midwives are used to advising women on potential health risks and nutrient deficiencies, but Samantha Nash argues that it is also important to consider one's own health.

Vitamin B12 is, to put it simply, ‘essential for life’ (Kenny and Tidy, 2016). Vitamin B12 deficiency can result in symptoms of fatigue, poor memory, pins and needles, mental health issues, difficulty conceiving, mobility and balance problems, and a plethora of haematological and neuropsychiatric problems (Lindenbaum et al, 1988; Hector and Burton, 1988; Savage and Lindenbaum, 1995; Bennett, 2001; Pacholok and Stuart, 2011; Sahoo et al, 2011; National Institute for Health and Care Excellence (NICE), 2015a; NHS Choices, 2016; Singh, 2016).

Prevalence of vitamin B12 deficiency has proven difficult to ascertain owing to the poor quality of available data. In the UK, a conservative estimate equates to approximately 6% of people under the age of 60 years, and closer to 20% for those aged 60 years and over (Hunt et al, 2014). Although the data available are of limited quality, the World Health Organization (2016) acknowledges that vitamin B12 deficiency could be a serious public health problem, potentially affecting millions worldwide.

Folic acid supplementation is recommended during pre-conception and in early pregnancy to reduce the risk of the fetus developing a neural tube defect. Jaquier (2015) suggests that 0.4 mg of folic acid reduces the risk of neural tube defects by 50–70%. These are impressive statistics, and underline the importance of supplementation for improving outcomes. However, practitioners should be aware that supplementation of folic acid can mask a vitamin B12 deficiency (NHS Choices, 2016). As is the case with low folate, low vitamin B12 can also cause neural tube defects, alongside miscarriage and premature birth (Pacholok and Stuart, 2011). NHS Choices (2015) advises that GPs should assess serum vitamin B12 before supplementing with folic acid—an intervention that would clearly be of benefit to those considering pregnancy.

In infants and children, vitamin B12 deficiency may manifest as difficulty with feeding—most notably sucking and swallowing—and failure to thrive. Such symptoms may be mistaken for tongue-tie (Pacholok and Stuart, 2011; Guez et al, 2012). Infants who are breastfed by a woman with an undiagnosed vitamin B12 deficiency may be at particular risk (Pacholok and Stuart, 2011). Molecular biologist Eva Greibe found that the level of vitamin B12 in breast milk may not remain optimal, as it declines over a period of 4 months (Fedders, 2013).

Vitamin B12 deficiency is completely inclusive of the human life span, affecting fetal development (Pacholok and Stuart, 2011; Pepper and Black, 2011; NHS Choices, 2016) through to the end of life, with dementia-type symptoms being a common manifestation (Savage and Lindenbaum, 1995; Lindenbaum et al, 2008; Pacholok and Stuart, 2011; NHS Choices, 2016). Some individuals are at increased risk of deficiency. Vegans and vegetarians can be particularly susceptible, as vitamin B12 is sourced entirely from animal produce i.e. meat, fish and dairy (Walsh, 2001; Herrmann and Obied, 2008; Pacholok and Stuart, 2011; NHS Choices, 2015). In addition, individuals who have undergone gastric bypass surgery, those with eating disorders or alcoholism, and people living with Crohn's and coeliac disease, are at increased risk (Pacholok and Stuart, 2011). Long-term use of metformin, a drug commonly used in pregnancy to control raised blood glucose levels for gestational diabetes (NICE, 2015b), can interrupt the metabolism of vitamin B12, resulting in a deficiency (de Jager et al, 2010). This highlights that a number of women in the care of midwives may be at risk of vitamin B12 deficiency.

A risk that is particularly relevant to midwifery is the use of nitrous oxide (Pacholok and Stuart, 2011; BOC Healthcare, 2015; 2016). In the UK, Entonox—the combination of nitrous oxide and oxygen—is often referred to as ‘gas and air’. It is generally considered a reasonably effective and safe analgesic. However, unlike other methods of medicine administration, inhalation of a gas has the potential to contaminate the working environment (BOC Healthcare, 2015; 2016; Royal College of Midwives, 2016). The manufacturer recommends that Entonox should be administered in adequately ventilated rooms (BOC Healthcare, 2015) and the UK Health and Safety Executive has set occupational exposure to a limit of 100 ppm over 8 hours (BOC Healthcare, 2015). Entonox should not be used for more than 24 hours, or more frequently than every 4 days (BOC Healthcare, 2016). Setting guidelines for health and safety is essential to protect the wellbeing of staff. However, there are some problems in adhering to these recommendations. Many midwives now work mandatory 12-hour shifts, and their shifts are often worked consecutively; therefore, midwives are invariably exposed to Entonox more frequently than the recommended 4-day interval (BOC Healthcare, 2016). A well-ventilated room is not always available, or even safe, especially when welcoming a new baby into the world where open windows or air conditioning increase the risk of cold injury to the newborn. Not all hospitals have scavenging systems and, even if they do, it could be argued that the gas would have to reach the scavenging system before reaching the people in the room. In addition, the Entonox recommendations do not appear to account for midwives attending homebirths, where the environment is out of the practitioner's control.

A common assumption may be that, as a gas, nitrous oxide is easily dispersed. However, oxygen is the lighter gas, and nitrous oxide collects in pockets, often at floor level (Piziali et al, 1976). Many midwives supporting an active birth may spend considerable time at floor level—sometimes hours. Even practitioners caring for women at bed level remain vulnerable. Normal movement in the room disturbs these pockets of nitrous oxide, exposing anyone in the vicinity to the gas and potentially increasing the risk of developing a vitamin B12 deficiency. An awareness of the risk is vital. Midwives should observe themselves and others for any subtle changes that could be indicative of a B12 deficiency.

My advice to midwives to protect themselves and their colleagues, along with the women and infants in their care, is to think ‘B12D’:

  • B: Be aware and look out for signs and symptoms of vitamin B12 deficiency. For a comprehensive list of signs and symptoms, see www.b12deficiency.info
  • 1: One-yearly screening to protect midwives would be advantageous. Early diagnosis and treatment reduces the risk of long-term complications, therefore benefiting the NHS by reducing the risk of staff ill health and the cost of sick leave. In pregnancy, serum B12 should be requested whenever a full blood count is required to assess for iron-deficiency anaemia i.e. at the booking appointment and 28-week contact. Serum B12 should be requested any time if a person becomes symptomatic for vitamin B12 deficiency. Practitioners should also be mindful that serum B12 is a poor indicator of vitamin B12 deficiency, as serum B12 can present as normal even when severely deficient (Devalia et al, 2014). Good practice would be to request either a plasma methylmalonic acid, homocysteine or serum holotranscobalamin (Devalia et al, 2014)
  • 2: Treat clinical symptoms, not levels of serum B12. Neuropsychiatric symptoms can be present without haematological changes and with an absence of anaemia (Lindenbaum et al, 1988; Devalia et al, 2014). Hydroxocobalamin, the parenteral treatment for vitamin B12 deficiency, is considered safe and non-toxic (Stichting Tekort, 2016)
  • D: Don't rely on supplements to treat deficiency. Oral vitamin B12 supplements, skin patches and sprays can skew serum B12 blood values by increasing haem atological values of vitamin B12 while allowing neuro psychiatric damage to advance (Dobson and Alvares, 2016).
  • Vitamin B12 deficiency can be devastating, but with early diagnosis and appropriate treatment a full recovery is possible. Individual holistic assessment of women and infants is essential to provide optimum care. This should include screening for vitamin B12 deficiency, and such screening should be extended to midwives when working around Entonox. When it comes to vitamin B12 deficiency, care and consideration should be given not only to women and infants, but to midwives and other health professionals.